Clearly structured and any time avai-lable case studies and presentations can be found at the VetContact site. The most recent contribution in the dermatology section has been written
by Dr. Stefanie Peters. „Congress Info“ is proud to offer the English version exclusively in this issue.
SIGNALEMENT: ROTTWEILER „CHUG“, 9 YEARS, MC
History:
„Chug“ has had severe hip dysplasia for many years and receives carprofen (Rimadyl®) p.o. from time to time. About 6 month ago, he showed spots of white hair over the whole body with-out any skin lesions. At the same time, the owners noticed a progressive depig-mentation of the planum nasale and intermittent epistaxis. The veterinarian diagnosed hypothyroidism and recom-mended T4 substitution because the dog was depressed. Additionally, „Chug“ was fed with Hill´s R/D because he was overweight.
For some weeks, „Chug“ was reluctant to walk and showed shifting lameness. Sloughing of the footpads, ulcerative lesions and „crackling paws“ were noted. An in vitro „allergy test“ was performed and showed a slight positive reaction to house dust and storage mites. Sarcoptes antibodies were negative. Because the veterinarian spotted multiple skin nodules, the dog was referred to a dermatologist.
The disease was neither contagious for the owners and for the 2 other dogs. The dog was born in Germany and had never been abroad.
|  | Clinical signs:
 1. Ulcera, sloughing footpads, „crackling footpads“ and depigmentation of all footpads.
 2. Depigmentation, erosions and some ulcerative lesions at all mucocutaneous junctions
 3. Depigmentation, erosions and some ulcerative lesions on the planum nasale with loss of the nasal architecture
|  |  4. Generalised multifocal leukotrichia and –derma
 5. Erythema, erosions and a few ulcers on tongue, gingiva and hard palate, multiple dermal plaques and nodules up to 2 cm size.
Differential diagnoses:
• Epitheliotropic T cell lymphoma („Mykosis fungoides“)
• „Drug eruption“ (Levothyroxin?, AB?, Rimadyl?)
• Leishmaniasis (not endemic in Germany)
• Pemphigus vulgaris, Bullous Pemphigoid, Pemphigus erythematosus, SLE
• Mucocutaneous pyoderma
• Vitiligo
The 3 first differentials were considered most likely because they could explain all the lesions that were noted.
Examinations performed:
• Cytology – not diagnostic (some cocci, degenerated PMN, some macrophages, few lymphocytes)
• Multiple biopsies for histopathological examination.
Histopathological results:
• Epidermis: acanthosis, orthokeratotic hyperkeratosis, ulceration, degene-rated subcorneal pustules with coocoid bacteria,
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