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Corynebacterium pseudotuberculosis outbreak causes skin lesions and mastitis
Mastitis is a well-known sign for a corynebacterium pseudotuberculosis infection. But this very interesting article from Israel describes a corynebacterium pseudotuberculosis outbreak in a dairy cattle herd causing both ulcerative granulomatous skin lesions and mastitis.

The disease was characterized by ulcerative granulomatous lesions, which occurred in an epidemic form.

Thirty-two cows and two heifers were affected, the ratio of the number affected to number at risk being 17.5 : 1 and 9.5 : 1, respectively.

The culling rate was 50% of the affected animals.

Most of the affected animals were cows (91.2%), with one first-calving cow (2.9%) and two heifers (5.9%) also affected.

The infection occurred during the summer to autumn months (August-December), and lasted 118 days.
The incubation period is about 2 months.

The disease appeared in two clinical forms cutaneous and mastitic or as a mixed form.

C. pseudotuberculosis organisms that were isolated from the ulcerative granulomatous lesions and from milk samples failed to reduce nitrate.

A decrease in milk production (4%) and an increase in the bulk-milk somatic cell count from a herd mean of 240 103 mL1 to 460 103 mL1 were noted during the morbidity period.

The organism was isolated from milk samples of eight animals (25%).

Source: YERUHAM, ISRAEL, FRIEDMAN, SAMUEL, PERL, SAMUEL, ELAD, DANIEL, BERKOVICH, YAIR & KALGARD, YORAM (2004) :A herd level analysis of a Corynebacterium pseudotuberculosis outbreak in a dairy cattle herd. In: Veterinary Dermatology 15 (5), 315-320.




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BOVINE

Zinc Deficiency-Like Syndrome in Fleckvieh Calvesmembers
Zinc deficiency-like (ZDL) syndrome is an inherited defect of Fleckvieh calves, with striking similarity to bovine hereditary zinc deficiency (BHZD). However, the causative mutation in a phospholipase D4 encoding gene (PLD4) shows no connection to zinc metabolism. The objectives of this study were to describe clinical signs, laboratory variables, and pathological findings of ZDL syndrome and their utility to differentiate ZDL from BHZD and infectious diseases with similar phenotype.

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