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Chronic wasting disease: New insights
Chronic wasting disease (CWD) is a unique transmissible spongiform encephalopathy (TSE) of mule deer (Odocoileus hemionus), white-tailed deer (O. virginianus), and Rocky Mountain elk (Cervus elaphus nelsoni). Still many facts regarding this disease remain unclear. A very interesting brandnew article...

The natural history of CWD is incompletely understood, but it differs from scrapie and bovine spongiform encephalopathy (BSE) by virtue of its occurrence in nondomestic and free-ranging species.

CWD has many features in common with scrapie, including early widespread distribution of disease-associated prion protein (PrPd) in lymphoid tissues, with later involvement of central nervous system (CNS) and peripheral tissues.

This distribution likely contributes to apparent efficiency of horizontal transmission and, in this, is similar to scrapie and differs from BSE. Clinical features and lesions of CWD are qualitatively similar to the other animal TSEs.

Microscopically, marked spongiform lesions occur in the central nervous system (CNS) after a prolonged incubation period and variable course of clinical disease.

During incubation, PrPd can be identified in tissues by antibody-based detection systems. Although CWD can be transmitted by intracerebral inoculation to cattle, sheep, and goats, ongoing studies have not demonstrated that domestic livestock are susceptible via oral exposure, the presumed natural route of exposure to TSEs.

Surveillance efforts for CWD in captive and free-ranging cervids will continue in concert with similar activities for scrapie and BSE.

Eradication of CWD in farmed cervids is the goal of state, federal, and industry programs, but eradication of CWD from free-ranging populations of cervids is unlikely with currently available management techniques.



Source: E. S. Williams (2005): Chronic Wasting Disease. In: Vet Pathol 42:530-549 (2005)



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BOVINE

Zinc Deficiency-Like Syndrome in Fleckvieh Calvesmembers
Zinc deficiency-like (ZDL) syndrome is an inherited defect of Fleckvieh calves, with striking similarity to bovine hereditary zinc deficiency (BHZD). However, the causative mutation in a phospholipase D4 encoding gene (PLD4) shows no connection to zinc metabolism. The objectives of this study were to describe clinical signs, laboratory variables, and pathological findings of ZDL syndrome and their utility to differentiate ZDL from BHZD and infectious diseases with similar phenotype.

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  • Tricho-dento-osseus-like syndrome in a Brown Swiss calfmembers
  • Non-tuberculous Mycobacteria in healthy slaughtered cattle and the abattoir members
  • Subclinical ketosis in dairy cows: prevalence and risk factorsmembers
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  • Antibodies to bovine viral diarrhoea virus (BVDV) in water buffalo and cattle in Australiamembers
  • Postpartum anoestrus in seasonally-calving dairy farms members


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